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[–]Nombre27 1 insightful - 1 fun1 insightful - 0 fun2 insightful - 1 fun -  (3 children)

[–]raven9 2 insightful - 1 fun2 insightful - 0 fun3 insightful - 1 fun -  (2 children)

Yes. You can also read it online here.

https://www.researchgate.net/publication/344407095_SARS-CoV-2_uses_CD4_to_infect_T_helper_lymphocytes

If you read the paper, aside from the fact they found the virus infects CD4+ cells the author states,

Remarkably, the same monoclonal antibody that has been used to block HIV entry in CD4+ T cells also blocked SARS-CoV-2 entry...

I am sure they knew well enough the implications of that. Here is what I believe could happen. When SARS-CoV-2 infects CD4+ T Lymphocytes it is untouchable. The immune system cannot turn on its own CD4 cells to destroy the virus within. The virus will therefore sit in those cells while they are inactive then when they are activated by other infections in future, the virus will start replicating again and it will infect more CD4 cells. This will happen every time we get so much as a cold. I therefore think the vast majority of us that were already exposed to the virus either symptomatically or asymptomatically are already the equivalent of HIV positive and no amount of mRNA spike proteins will change that while the virus resides within our own immune system.

[–]Nombre27 2 insightful - 1 fun2 insightful - 0 fun3 insightful - 1 fun -  (1 child)

That sounds like a horror movie. I hope you're wrong.

Does SARS-CoV-2 remain dormant in the CD4+ cells though?

[–]raven9 2 insightful - 1 fun2 insightful - 0 fun3 insightful - 1 fun -  (0 children)

Well that's the thing, no one has published any further research into it since that paper from Brazil. I think it is similar to 911 when nothing was allowed to contradict the official narrative. Who will fund research knowing it will not be published and that they will probably be blackballed for doing it? I think we were lucky to have seen as much as we have.